ARDS(급성호흡곤란증후군)

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"ARDS(급성호흡곤란증후군)"에 대한 내용입니다.

목차

1. Clinical Principles of ARDS
2. Physiologic Principles of ARDS
3. Pulmonary Edema
4. ARDS
5. Etiology of ARDS
6. Risk Factors (Host Factors)
7. Why So Important is the ARDS ?
8. Introduction
9. The American-European Consensus Conference Definition
10. Clinical-Pathologic Correlation In ARDS
11. Causes of ARDS
12. Physiologic Basis of ARDS
13. Physiologic Basis of Pulmonary Edema
14. Why Does Edema Accumulate in ARDS ?
15. Inflammation Activates Capillary Endothelium in ARDS
16. Lung Protective Ventilation Strategies in ARDS
17. Treatment of the ARDS
18. Prognosis of the ARDS
19. Summary

본문내용

Clinical Principles of ARDS
•Severe resp. distress & > 1 risk factors :
–Infection, aspiration, pancreatitis, trauma
•Impaired arterial oxygenation (hypoxemia)
•Bilateral pulm. infitration on chest X-ray
•No clinical evidence of ↑ LA pressure
–PAWP ≤ 18 mmHg

Physiologic Principles of ARDS
•Refractory hypoxemia is caused by formation of protein-rich alveolar edema d/t damage to the integrity of the alveolar-capillary barrier.
•Alveolar-capillary damage can be initiated by physical or chemical injury or by extensive activation of innate inflammatory responses.

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